Rodger Garrett
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I have run across other material in the PTSD literature suggesting that overproduction and/or over-long production of pro-inflammatory cytokines is a principle factor in the excitotoxicty of dendritic receptor sites in the limbic system of PTSD sufferers. I am utilizing this state-of-the-art-in-2005 description of what was known at that time about the functions of the hypothalamo-pituitary-adrenal (HPA) axis and the sympathetic nervous system (SNS) as a platform to introduce what I have learned elsewhere about excitotoxity. The purpose is to provide an explanation of the micro-neurobiology of PTSD as I have been able to see it in the behavior and computer-adied tomography of PTSD sufferers. I would like to invite any and all who have superior grasp of the topic to weigh in with comments, factual corrections, etc.
I am indebted to Dr. Molina and to Lippincott, Williams and Williams for making the original article available for this usage. You may see the article and commentary by contacting me through the forum.
I am indebted to Dr. Molina and to Lippincott, Williams and Williams for making the original article available for this usage. You may see the article and commentary by contacting me through the forum.